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Observations on the structure of the entozoa belonging to the genus cysticercus.

Medico-chirurgical transactions

Gulliver G.
PMID: 20895723
Med Chir Trans. 1841;24:1-250.2. doi: 10.1177/095952874102400102.

No abstract available.

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Gulliver G. Observations on the structure of the entozoa belonging to the genus cysticercus. Med Chir Trans. 1841;24:1-250.2doi: 10.1177/095952874102400102.
Gulliver, G. (1841). Observations on the structure of the entozoa belonging to the genus cysticercus. Medico-chirurgical transactions, 241-250.2. https://doi.org/10.1177/095952874102400102
Gulliver, G. "Observations on the structure of the entozoa belonging to the genus cysticercus." Medico-chirurgical transactions vol. 24 (1841): 1-250.2. doi: https://doi.org/10.1177/095952874102400102
Gulliver G. Observations on the structure of the entozoa belonging to the genus cysticercus. Med Chir Trans. 1841;24:1-250.2. doi: 10.1177/095952874102400102. PMID: 20895723; PMCID: PMC2116753.
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Innate immune signaling and gut-liver interactions in non-alcoholic fatty liver disease.

Hepatobiliary surgery and nutrition

Bieghs V, Trautwein C.
PMID: 25568861
Hepatobiliary Surg Nutr. 2014 Dec;3(6):377-85. doi: 10.3978/j.issn.2304-3881.2014.12.04.

Non-alcoholic fatty liver disease (NAFLD) is the hepatic manifestation of the metabolic syndrome and covers a disease spectrum ranging from steatosis to inflammation, fibrosis, cirrhosis and hepatocellular carcinoma (HCC). The innate immune response in the liver plays an important...

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Bieghs V, Trautwein C. Innate immune signaling and gut-liver interactions in non-alcoholic fatty liver disease. Hepatobiliary Surg Nutr. 2014;3(6):377-85doi: 10.3978/j.issn.2304-3881.2014.12.04.
Bieghs, V., & Trautwein, C. (2014). Innate immune signaling and gut-liver interactions in non-alcoholic fatty liver disease. Hepatobiliary surgery and nutrition, 3(6), 377-85. https://doi.org/10.3978/j.issn.2304-3881.2014.12.04
Bieghs, Veerle, and Trautwein, Christian. "Innate immune signaling and gut-liver interactions in non-alcoholic fatty liver disease." Hepatobiliary surgery and nutrition vol. 3,6 (2014): 377-85. doi: https://doi.org/10.3978/j.issn.2304-3881.2014.12.04
Bieghs V, Trautwein C. Innate immune signaling and gut-liver interactions in non-alcoholic fatty liver disease. Hepatobiliary Surg Nutr. 2014 Dec;3(6):377-85. doi: 10.3978/j.issn.2304-3881.2014.12.04. PMID: 25568861; PMCID: PMC4273113.
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Helicobacter pylori Infection Enhances Gastric Mucosal Inflammation in Individuals Carrying the 260-T Allele of the CD14 Gene.

Gut and liver

Kim EJ, Chung WC, Lee KM, Paik CN, Kim SB, Oh YS, Lee YW, Kang SG, Noh SJ.
PMID: 23710313
Gut Liver. 2013 May;7(3):317-22. doi: 10.5009/gnl.2013.7.3.317. Epub 2013 May 13.

BACKGROUND/AIMS: We aim to evaluate the association between promoter polymorphism of the clusters of differentiation 14 (CD14) gene and Helicobacter pylori-induced gastric mucosal inflammation in a healthy Korean population.METHODS: The study population consisted of 267 healthy subjects who visited...

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Kim EJ, Chung WC, Lee KM, et al. Helicobacter pylori Infection Enhances Gastric Mucosal Inflammation in Individuals Carrying the 260-T Allele of the CD14 Gene. Gut Liver. 2013;7(3):317-22doi: 10.5009/gnl.2013.7.3.317.
Kim, E. J., Chung, W. C., Lee, K. M., Paik, C. N., Kim, S. B., Oh, Y. S., Lee, Y. W., Kang, S. G., & Noh, S. J. (2013). Helicobacter pylori Infection Enhances Gastric Mucosal Inflammation in Individuals Carrying the 260-T Allele of the CD14 Gene. Gut and liver, 7(3), 317-22. https://doi.org/10.5009/gnl.2013.7.3.317
Kim, Eun Jung, et al. "Helicobacter pylori Infection Enhances Gastric Mucosal Inflammation in Individuals Carrying the 260-T Allele of the CD14 Gene." Gut and liver vol. 7,3 (2013): 317-22. doi: https://doi.org/10.5009/gnl.2013.7.3.317
Kim EJ, Chung WC, Lee KM, Paik CN, Kim SB, Oh YS, Lee YW, Kang SG, Noh SJ. Helicobacter pylori Infection Enhances Gastric Mucosal Inflammation in Individuals Carrying the 260-T Allele of the CD14 Gene. Gut Liver. 2013 May;7(3):317-22. doi: 10.5009/gnl.2013.7.3.317. Epub 2013 May 13. PMID: 23710313; PMCID: PMC3661964.
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Starring role of toll-like receptor-4 activation in the gut-liver axis.

World journal of gastrointestinal pathophysiology

Carotti S, Guarino MP, Vespasiani-Gentilucci U, Morini S.
PMID: 26600967
World J Gastrointest Pathophysiol. 2015 Nov 15;6(4):99-109. doi: 10.4291/wjgp.v6.i4.99.

Since the introduction of the term "gut-liver axis", many studies have focused on the functional links of intestinal microbiota, barrier function and immune responses to liver physiology. Intestinal and extra-intestinal diseases alter microbiota composition and lead to dysbiosis, which...

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Carotti S, Guarino MP, Vespasiani-Gentilucci U, et al. Starring role of toll-like receptor-4 activation in the gut-liver axis. World J Gastrointest Pathophysiol. 2015;6(4):99-109doi: 10.4291/wjgp.v6.i4.99.
Carotti, S., Guarino, M. P., Vespasiani-Gentilucci, U., & Morini, S. (2015). Starring role of toll-like receptor-4 activation in the gut-liver axis. World journal of gastrointestinal pathophysiology, 6(4), 99-109. https://doi.org/10.4291/wjgp.v6.i4.99
Carotti, Simone, et al. "Starring role of toll-like receptor-4 activation in the gut-liver axis." World journal of gastrointestinal pathophysiology vol. 6,4 (2015): 99-109. doi: https://doi.org/10.4291/wjgp.v6.i4.99
Carotti S, Guarino MP, Vespasiani-Gentilucci U, Morini S. Starring role of toll-like receptor-4 activation in the gut-liver axis. World J Gastrointest Pathophysiol. 2015 Nov 15;6(4):99-109. doi: 10.4291/wjgp.v6.i4.99. PMID: 26600967; PMCID: PMC4644892.
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Gut Microbiota and Hepatocellular Carcinoma.

Gastrointestinal tumors

Tao X, Wang N, Qin W.
PMID: 26673641
Gastrointest Tumors. 2015 May;2(1):33-40. doi: 10.1159/000380895. Epub 2015 Apr 22.

BACKGROUND: Hepatocellular carcinoma (HCC) is a common complication of liver diseases such as those related to viral hepatitis and liver cirrhosis. The gut-liver axis is gaining increasing attention as a key pathophysiological mechanism responsible for the progression of HCC....

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Tao X, Wang N, Qin W. Gut Microbiota and Hepatocellular Carcinoma. Gastrointest Tumors. 2015;2(1):33-40doi: 10.1159/000380895.
Tao, X., Wang, N., & Qin, W. (2015). Gut Microbiota and Hepatocellular Carcinoma. Gastrointestinal tumors, 2(1), 33-40. https://doi.org/10.1159/000380895
Tao, Xueme, et al. "Gut Microbiota and Hepatocellular Carcinoma." Gastrointestinal tumors vol. 2,1 (2015): 33-40. doi: https://doi.org/10.1159/000380895
Tao X, Wang N, Qin W. Gut Microbiota and Hepatocellular Carcinoma. Gastrointest Tumors. 2015 May;2(1):33-40. doi: 10.1159/000380895. Epub 2015 Apr 22. PMID: 26673641; PMCID: PMC4668791.
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Joint Transmit Antenna Selection and Power Allocation for ISDF Relaying Mobile-to-Mobile Sensor Networks.

Sensors (Basel, Switzerland)

Xu L, Zhang H, Gulliver TA.
PMID: 26907282
Sensors (Basel). 2016 Feb 19;16(2):249. doi: 10.3390/s16020249.

The outage probability (OP) performance of multiple-relay incremental-selective decode-and-forward (ISDF) relaying mobile-to-mobile (M2M) sensor networks with transmit antenna selection (TAS) over N-Nakagami fading channels is investigated. Exact closed-form OP expressions for both optimal and suboptimal TAS schemes are derived....

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Xu L, Zhang H, Gulliver TA. Joint Transmit Antenna Selection and Power Allocation for ISDF Relaying Mobile-to-Mobile Sensor Networks. Sensors (Basel). 2016;16(2):249doi: 10.3390/s16020249.
Xu, L., Zhang, H., & Gulliver, T. A. (2016). Joint Transmit Antenna Selection and Power Allocation for ISDF Relaying Mobile-to-Mobile Sensor Networks. Sensors (Basel, Switzerland), 16(2), 249. https://doi.org/10.3390/s16020249
Xu, Lingwei, et al. "Joint Transmit Antenna Selection and Power Allocation for ISDF Relaying Mobile-to-Mobile Sensor Networks." Sensors (Basel, Switzerland) vol. 16,2 (2016): 249. doi: https://doi.org/10.3390/s16020249
Xu L, Zhang H, Gulliver TA. Joint Transmit Antenna Selection and Power Allocation for ISDF Relaying Mobile-to-Mobile Sensor Networks. Sensors (Basel). 2016 Feb 19;16(2):249. doi: 10.3390/s16020249. PMID: 26907282; PMCID: PMC4801625.
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Draft Genome Sequence of .

Genome announcements

Lipus D, Ross D, Bibby K, Gulliver D.
PMID: 28302780
Genome Announc. 2017 Mar 16;5(11). doi: 10.1128/genomeA.00033-17.

We report the 5,425,832 bp draft genome of

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Lipus D, Ross D, Bibby K, et al. Draft Genome Sequence of . Genome Announc. 2017;5(11)doi: 10.1128/genomeA.00033-17.
Lipus, D., Ross, D., Bibby, K., & Gulliver, D. (2017). Draft Genome Sequence of . Genome announcements, 5(11), . https://doi.org/10.1128/genomeA.00033-17
Lipus, Daniel, et al. "Draft Genome Sequence of ." Genome announcements vol. 5,11 (2017). doi: https://doi.org/10.1128/genomeA.00033-17
Lipus D, Ross D, Bibby K, Gulliver D. Draft Genome Sequence of . Genome Announc. 2017 Mar 16;5(11). doi: 10.1128/genomeA.00033-17. PMID: 28302780; PMCID: PMC5356057.
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Gastroesophageal reflux disease and tooth erosion: a cross-sectional observational study.

Gut and liver

Farahmand F, Sabbaghian M, Ghodousi S, Seddighoraee N, Abbasi M.
PMID: 23710307
Gut Liver. 2013 May;7(3):278-81. doi: 10.5009/gnl.2013.7.3.278. Epub 2013 Apr 09.

BACKGROUND/AIMS: Gastroesophageal reflux disease (GERD) is common in children. Recurrent exposure to gastric acid in GERD may contribute to tooth erosion.METHODS: In this prospective study, 54 GERD patients qualified according to endoscopy, pH-metry, and the GERD questionnaire and 58...

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Farahmand F, Sabbaghian M, Ghodousi S, et al. Gastroesophageal reflux disease and tooth erosion: a cross-sectional observational study. Gut Liver. 2013;7(3):278-81doi: 10.5009/gnl.2013.7.3.278.
Farahmand, F., Sabbaghian, M., Ghodousi, S., Seddighoraee, N., & Abbasi, M. (2013). Gastroesophageal reflux disease and tooth erosion: a cross-sectional observational study. Gut and liver, 7(3), 278-81. https://doi.org/10.5009/gnl.2013.7.3.278
Farahmand, Fatemeh, et al. "Gastroesophageal reflux disease and tooth erosion: a cross-sectional observational study." Gut and liver vol. 7,3 (2013): 278-81. doi: https://doi.org/10.5009/gnl.2013.7.3.278
Farahmand F, Sabbaghian M, Ghodousi S, Seddighoraee N, Abbasi M. Gastroesophageal reflux disease and tooth erosion: a cross-sectional observational study. Gut Liver. 2013 May;7(3):278-81. doi: 10.5009/gnl.2013.7.3.278. Epub 2013 Apr 09. PMID: 23710307; PMCID: PMC3661958.
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Prediction of Advanced Fibrosis in Nonalcoholic Fatty Liver Disease: An Enhanced Model of BARD Score.

Gut and liver

Lee TH, Han SH, Yang JD, Kim D, Ahmed M.
PMID: 23710314
Gut Liver. 2013 May;7(3):323-8. doi: 10.5009/gnl.2013.7.3.323. Epub 2013 Apr 25.

BACKGROUND/AIMS: The BARD score is a model to detect advanced liver fibrosis in nonalcoholic fatty liver disease (NAFLD) patients. The aims of this study were to identify additional factors and then to build an enhanced version of the BARD...

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Lee TH, Han SH, Yang JD, et al. Prediction of Advanced Fibrosis in Nonalcoholic Fatty Liver Disease: An Enhanced Model of BARD Score. Gut Liver. 2013;7(3):323-8doi: 10.5009/gnl.2013.7.3.323.
Lee, T. H., Han, S. H., Yang, J. D., Kim, D., & Ahmed, M. (2013). Prediction of Advanced Fibrosis in Nonalcoholic Fatty Liver Disease: An Enhanced Model of BARD Score. Gut and liver, 7(3), 323-8. https://doi.org/10.5009/gnl.2013.7.3.323
Lee, Tae Hoon, et al. "Prediction of Advanced Fibrosis in Nonalcoholic Fatty Liver Disease: An Enhanced Model of BARD Score." Gut and liver vol. 7,3 (2013): 323-8. doi: https://doi.org/10.5009/gnl.2013.7.3.323
Lee TH, Han SH, Yang JD, Kim D, Ahmed M. Prediction of Advanced Fibrosis in Nonalcoholic Fatty Liver Disease: An Enhanced Model of BARD Score. Gut Liver. 2013 May;7(3):323-8. doi: 10.5009/gnl.2013.7.3.323. Epub 2013 Apr 25. PMID: 23710314; PMCID: PMC3661965.
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A Case of Common Bile Duct Cancer That Completely Responded to Combination Chemotherapy of Gemcitabine and TS-1.

Gut and liver

Lim JH, Ryu JK, Choi YJ, Kwon J, Kim JY, Lee YB, Kim JH, Yoon WJ, Kim YT, Yoon YB.
PMID: 23710321
Gut Liver. 2013 May;7(3):371-6. doi: 10.5009/gnl.2013.7.3.371. Epub 2013 May 13.

Common bile duct (CBD) cancer is a relatively rare malignancy that arises from the biliary epithelium and is associated with a poor prognosis. Here, we report a case of advanced metastatic CBD cancer successfully treated by chemotherapy with gemcitabine...

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Lim JH, Ryu JK, Choi YJ, et al. A Case of Common Bile Duct Cancer That Completely Responded to Combination Chemotherapy of Gemcitabine and TS-1. Gut Liver. 2013;7(3):371-6doi: 10.5009/gnl.2013.7.3.371.
Lim, J. H., Ryu, J. K., Choi, Y. J., Kwon, J., Kim, J. Y., Lee, Y. B., Kim, J. H., Yoon, W. J., Kim, Y. T., & Yoon, Y. B. (2013). A Case of Common Bile Duct Cancer That Completely Responded to Combination Chemotherapy of Gemcitabine and TS-1. Gut and liver, 7(3), 371-6. https://doi.org/10.5009/gnl.2013.7.3.371
Lim, Joo Hyun, et al. "A Case of Common Bile Duct Cancer That Completely Responded to Combination Chemotherapy of Gemcitabine and TS-1." Gut and liver vol. 7,3 (2013): 371-6. doi: https://doi.org/10.5009/gnl.2013.7.3.371
Lim JH, Ryu JK, Choi YJ, Kwon J, Kim JY, Lee YB, Kim JH, Yoon WJ, Kim YT, Yoon YB. A Case of Common Bile Duct Cancer That Completely Responded to Combination Chemotherapy of Gemcitabine and TS-1. Gut Liver. 2013 May;7(3):371-6. doi: 10.5009/gnl.2013.7.3.371. Epub 2013 May 13. PMID: 23710321; PMCID: PMC3661972.
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Endoscopic ultrasound-guided therapeutic thoracentesis.

Gut and liver

Luz LP, El Hajj II, Dewitt JM.
PMID: 23710323
Gut Liver. 2013 May;7(3):382-3. doi: 10.5009/gnl.2013.7.3.382. Epub 2013 May 13.

No abstract available.

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Luz LP, El Hajj II, Dewitt JM. Endoscopic ultrasound-guided therapeutic thoracentesis. Gut Liver. 2013;7(3):382-3doi: 10.5009/gnl.2013.7.3.382.
Luz, L. P., El Hajj, I. I., & Dewitt, J. M. (2013). Endoscopic ultrasound-guided therapeutic thoracentesis. Gut and liver, 7(3), 382-3. https://doi.org/10.5009/gnl.2013.7.3.382
Luz, Leticia P, et al. "Endoscopic ultrasound-guided therapeutic thoracentesis." Gut and liver vol. 7,3 (2013): 382-3. doi: https://doi.org/10.5009/gnl.2013.7.3.382
Luz LP, El Hajj II, Dewitt JM. Endoscopic ultrasound-guided therapeutic thoracentesis. Gut Liver. 2013 May;7(3):382-3. doi: 10.5009/gnl.2013.7.3.382. Epub 2013 May 13. PMID: 23710323; PMCID: PMC3661974.
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PATHOGENESIS OF HEPATOCELLULAR CARCINOMA DEVELOPMENT IN NON ALCOHOLIC FATTY LIVER DISEASE.

Current hepatology reports

Shetty K, Chen J, Shin JH, Jogunoori W, Mishra L.
PMID: 26114083
Curr Hepatol Rep. 2015 Jun;14(2):119-127. doi: 10.1007/s11901-015-0260-z.

Non-alcoholic fatty liver disease (NAFLD) is being recognized as an increasingly important contributor to the burden of hepatocellular carcinoma (HCC) worldwide. It is often accompanied by obesity and diabetes mellitus and is believed to be the hepatic representation of...

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Shetty K, Chen J, Shin JH, et al. PATHOGENESIS OF HEPATOCELLULAR CARCINOMA DEVELOPMENT IN NON ALCOHOLIC FATTY LIVER DISEASE. Curr Hepatol Rep. 2015;14(2):119-127doi: 10.1007/s11901-015-0260-z.
Shetty, K., Chen, J., Shin, J. H., Jogunoori, W., & Mishra, L. (2015). PATHOGENESIS OF HEPATOCELLULAR CARCINOMA DEVELOPMENT IN NON ALCOHOLIC FATTY LIVER DISEASE. Current hepatology reports, 14(2), 119-127. https://doi.org/10.1007/s11901-015-0260-z
Shetty, Kirti, et al. "PATHOGENESIS OF HEPATOCELLULAR CARCINOMA DEVELOPMENT IN NON ALCOHOLIC FATTY LIVER DISEASE." Current hepatology reports vol. 14,2 (2015): 119-127. doi: https://doi.org/10.1007/s11901-015-0260-z
Shetty K, Chen J, Shin JH, Jogunoori W, Mishra L. PATHOGENESIS OF HEPATOCELLULAR CARCINOMA DEVELOPMENT IN NON ALCOHOLIC FATTY LIVER DISEASE. Curr Hepatol Rep. 2015 Jun;14(2):119-127. doi: 10.1007/s11901-015-0260-z. PMID: 26114083; PMCID: PMC4476538.
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Showing 1 to 12 of 1026 entries